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Genetic polymorphisms in key components of vitamin D metabolism and signaling, including VDR, GC (vitamin D binding protein), CYP2R1, and CYP27B1, have been associated with differences in circulating 25(OH)D concentrations and downstream biological effects

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303 Upon exposure to multiple stresses, dysfunctional p53 can prevent the occurrence of atherosclerosis or promote the development of atherosclerosis in distinct cell types through p53-dependent apoptosis and the growth arrest response