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Exogenous GSH supplementation restores mitochondrial respiration and attenuates myocardial injury associated with diabetes (Tocchetti et al., 2012)
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BRCA1 can interact with NRF2, and this prohibits binding and ubiquitination by KEAP1 (230, 239, 240), thus making NRF2 stable and causing downstream activation of target genes and the anti-stress response
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