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Hepatotoxicity of di-(2-ethylhexyl)phthalate is attributed to calcium aggravation, ROS-mediated mitochondrial depolarization, and ERK/NF-kappaB pathway activation
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Researchers measure lesion area via endoscopic or gross pathology examination, alongside molecular markers of inflammation such as TNF-alpha, IL-1 beta, and COX-2 expression in gastric tissue samples
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