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Moreover, in animal models, ALC has been shown to attenuate the release of pro-inflammatory mediators (IL-1β, TNFα) from activated microglia, to modulate neuroinflammation and to reduce the concentration of oxidative stress markers, increasing the concentration of glutathione (GSH), a powerful mediator with antioxidant proprieties [27,28]
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Following that, GnRH agonist desensitizes the receptor, causing a decrease in gonadotropin release, and subsequently reduces the estrogen level secondary to the decrease in ovary stimulation by gonadotropin
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