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2A), studies have shown that abnormal FAO can not only directly affect drug resistance of cancer cells through metabolic reprogramming, but also by altering signal pathways, inhibiting autophagy of tumor cells and regulating the cell cycle [24, 51, 52]

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As a consequence of this, both the concentration and the permanence of glutamate in the synaptic cleft would be increased, potentiating the action of this neurotransmitter on the glutamatergic system (Fig