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There's a lot of evidence that in Alzheimer's, Parkinson's, definitely stroke, traumatic brain injury, ALS, all of those excitotoxicity is a factor that is neurons continuing to be excited by glutamate when they're in a compromised state, energetically, for example, with a stroke, or we think more subtly in Parkinson's or Alzheimer's, where there's mitochondrial dysfunction that's occurring
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The key findings from this clinical program include significant reductions in body weight and fat mass in treatment groups compared to placebo, preferential loss of abdominal and visceral adipose tissue, no changes in serum IGF-1 levels at any dose tested, no negative effects on glucose metabolism or insulin sensitivity, no significant adverse events related to metabolic or endocrine disruption, and safety profile indistinguishable from placebo for most parameters
