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Through bioactivity-guided chromatography and molecular experiments, HON was identified as a Transient receptor potential vanilloid 4 (TRPV4) antagonist, targeting the Q239 residue to inhibit channel opening and improve endothelial permeability [489]
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Colorectal cancer exhibits oxaliplatin resistance mediated by Fusobacterium nucleatum via the E-cadherin/β-catenin/TCF4/GPX4 axis, and the KIF20A/NUAK1/PP1β/GPX4 pathway may further support this resistance [61,62]
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