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Increased oxidative stress is also a key factor associated with aging and there is convincing evidence that Nrf2, not only declines with age, but that Nrf2 and HO-1 can reduce cellular senescence and the senescence-associated secretory phenotype (SASP) which is now considered an underlying driver of age-related inflammatory disease

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Uso Exclusivo em Pesquisa — Aviso Regulatório Todos os produtos e informações apresentados neste site destinam-se exclusivamente à pesquisa laboratorial in vitro e à investigação científica por pesquisadores qualificados

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