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Pharmacodynamics of S-dimethylarsino-glutathione, a putative metabolic intermediate of inorganic arsenic, in mice - ScienceDirect
Abolishing UCHL1's hydrolase activity exacerbates TBI-induced axonal injury and neuronal death in mice
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The effect was due to accelerated angiogenesis but not vasodilation, since there was no significant difference of the blood flow in tails and non-ischemic hind limb between the control and BPC 157 groups
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